Written in English
|Statement||by Brian Craig Prince.|
|The Physical Object|
|Pagination||viii, 48 leaves, bound ;|
|Number of Pages||48|
When administered systemically, oxytocin (OT) stimulates secretion of uterine prostaglandin F 2α (PGF 2α) in swine, but the role of endometrially-derived OT in control of PGF 2α release is not clear. This study determined the effect of exogenous OT, administered into the uterine lumen of intact cyclic gilts, on PGF 2α secretion during late diestrus.. Intrauterine infusion of 40 USP Cited by: 5. Exogenous OT decreases interestrous interval when administered to cyclic gilts between Days 10 and 16 post-estrus. The endometrium of pigs contains receptors for OT and lysine vasopressin, but only responds to OT with increased secretion of PGF [ ]. Moreover, exogenous OT, systemically administered to cyclic gilts, reduced interestrous intervals by – days (Sample et al., ). Oxytocin-stimulated endometrial secretion of prostaglandin F 2 α (PGF 2 α) in endometrium isolated from sows on days 15–16 of the oestrous cycle (Whiteaker et al., , Uzumcu et al., , Carnahan Cited by: Pregnant OT-treated gilts had increased (p cyclic gilts on Days 10 but lower than those.
The effect of the endotoxin injection on the irregularity structure of HRF series was also investigated using DFA and SampEn, depicted in Fig. short-term scaling exponent α 1 remained decreased (Fig. 2a) from + 7 to + 9 h post-LPS injection according to the results of planned AUC comparisons (F = ; p. W C Becker's 7 research works with citations and reads, including: Exogenous oxytocin decreases interestrous interval of cyclic gilts. Exogenous OT decreases interestrous interval when administered to cyclic gilts between days 10 and 16 post-estrus, but not when administered to ovarian- intact hysterectomized gilts, suggesting that the effect of OT is uterine-dependent (Mirando et al., ). Admin- istration of oxytocin stimulates endometrial release of PGf-za and shortens the interestrous interval of the ewe. Active and passive immunization of cyclic ewes against oxytocin or a continuous infusion of oxytocin delays luteal regression.
oxytocin for either induction or augmentation of labour.4 Although various oxytocin regimens for the induction or augmentation of labour have been described,5–9 relatively few studies have focused on the duration of oxytocin administration in labour–15 The use of oxytocin . Exogenous AVP in the anterior hypothalamus can stimulate offensive aggression [, ], but this effect may be modulated by social environment. Further work in hamsters has revealed that an orally active V1a antagonist decreases aggression in male hamsters, but does not affect social investigation or sexual motivation [ ]. published studies on the effects of exogenous oxytocin on social cognition and prosocial behavior in humans [1,2] motivated largely by research showing that oxytocin is involved in regulating such social processes in animals . The excitement about oxytocin has not been conﬁned tothescientiﬁccommunity:dubbedthe‘lovehormone’(e.g. Oxytocin (Oxt) is a peptide hormone and is normally produced in the hypothalamus and released by the posterior pituitary. It plays a role in social bonding, reproduction, childbirth, and the period after childbirth. Oxytocin is released into the bloodstream as a hormone in response to love and in labor.